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Hansson, J

91 Thus, GSH can drive efflux-mediated resistance by both enabling antibiotic efflux and modulating the redox state of efflux pump regulators

Identification in synovial fluid of a new potential pathogenic player in arthropathies

when iodine is lacking, under the stimulation of high TSH, thyroid cells produce more H2O2, whose accumulation gradually damages thyroid cells

To address this, we used a series of orthogonal approaches to test the role of NAD(P) + homeostasis in chemotherapy induced infertility, including treatment with the NAD + precursor NMN, along with transgenic overexpression of the NAD + biosynthetic enzymes nicotinamide mononucleotide adenyltransferase 1 and 3 (NMNAT1 and NMNAT3)