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Firstly, excessive reactive oxygen species (ROS) generation activates proinflammatory cascades, exacerbating ECM deposition and tubular injury

Overall, Se nutrition and selenoprotein activity have the potential to improve inflammatory response outcomes in several ways including combating oxidative stress in cells/tissues and through the redox regulation of inflammatory signalling pathways that lead to cytokine/chemokine production

Orr reports grants from Alzheimers Drug Discovery Foundation, grants from NIA, grants from NINDS, grants from US Department of Veterans Affairs, grants from Cure Alzheimers Fund, grants from AFAR/Hevolution during the conduct of the study