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We now know more and more about how this cycle can cause a wide range of conditions and impact our overall health

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Because GPX4 ablation or inaction is embryonically lethal, only heterozygous or conditional knockout mice can be analyzed (Table 1)

Hyperoxalemia leads to oxidative stress in endothelial cells and mice with chronic kidney disease

Grx2 knockout or overexpression exacerbates or diminishes lipid peroxidation in human lens epithelial cells or opacity in mouse lenses, respectively, after treatment with ferroptosis inducers or UV-B via modulation of mitochondrial structure and the GSH/GSSG ratio [128]