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L-carnitine facilitates the transport of long-chain fatty acids into the mitochondrial matrix, triggering cardioprotective effects through reduced oxidative stress, inflammation, and necrosis of cardiac myocytes

However, many current studies examining the relationship between RA, oxidative stress, and inflammation seem to share the conclusion that getting those factors under control is critical to stopping the arterial stiffness and damage of the disease

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Recently, it was found that the Gram-negative bacterium Pseudomonas aeruginosa (PA) can lead to the loss of intracellular GPX4 after infection of the host, resulting in ferroptosis [46]