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The amount of BA ingested that is converted into muscle carnosine is only about 36% (47), while small amounts (~3%) are also lost via the urine ( It appears reasonable to expect that any changes in muscle carnosine content would be paralleled by changes in the proteins involved in its metabolism
Some variants have clear structural explanations for their loss-of-function, for example, variants can disrupt binding of carnitine (R471C/H/P), affect the Na + -binding site (N32S), disrupt the TM2-TM11 H-bonding network (S467C) or perturb gating residues conserved in OCTs/OATs (R169Q/P/W)
Tablets are another popular format, often combined with supportive nutrients like vitamin C or alpha lipoic acid
doi: 10.3892/etm.2019.8388 316 LiGMaXDengLZhaoXWeiYGaoZet al
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