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The pathophysiology of TN is multifactorial and involves genetic predisposition, anatomical changes, and neurophysiological factors, leading to hyperexcitable neuronal states, central sensitization and widespread neural plasticity changes

Phase 3 trials are ongoing, and without completed large-scale safety and efficacy data, the compound cannot be considered ready for routine clinical use

Its role in insulin sensitivity has made it a subject of particular interest for patients with metabolic conditions

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