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Codon usage and mRNA stability are translational determinants of cellular response to canonical ferroptosis inducers

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Changes in deacetylase levels throughout aging may exacerbate the aging phenotype by impairing the kidneys' ability to counteract oxidative stress, potentially resulting in heightened oxidative DNA damage (Radak et al., 2013

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These in vivo results align with the in vitro data, consolidating the understanding that GHK-Cu exerts its protective effects in ALI by inhibiting excessive inflammatory responses through its anti-inflammatory and antioxidant properties, mediated by the suppression of NF-B and p38 MAPK signaling pathways [2]