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This suggests that APDs act directly on islet -cells, in addition to -cells, to disrupt the D 2 -like receptor signaling responsible for regulating islet hormone secretion

It also increased acetylcholinesterase (AChE) and butyrylcholinesterase (BuChE) activity, key enzymes that modulate acetylcholine, the neurotransmitter crucial for learning and memory [1]

TB4 lowered oxidative stress and lipid peroxidation, boosted antioxidants, suppressed inflammation by inhibiting nuclear factor kappa B, and prevented liver fibrosis [38]

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Also, see the Drug Facts panel(s) on the package