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A common pathway affecting all these risk factors involves a low measure of reduced glutathione (i.e., GSH) level (Khanfar and Al Qaroot, 2020

Anti-angiogenic drugs (VEGF pathway inhibitors and TKI) Mechanisms and clinical manifestations The premise of this type of targeted drugs is that tumor growth is highly dependent on local pathological angiogenesis induced by vascular endothelial growth factor (VEGF), which promotes tumor development by regulating vascular permeability, endothelial cell migration, proliferation, and survival (44)

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Uric acid shown to contribute to increased oxidative stress level independent of Xanthine oxidoreductase activity in MedCity21 health examination registry

The procognitive and synaptogenic effects of angiotensin IV-derived peptides are dependent on activation of the hepatocyte growth factor/c-Met system