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Our findings are in line with other observations suggesting that the ligature induced experimental periodontitis is a useful and highly reproducible model for gingival/periodontal inflammation (41, 42)

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Preclinical PTSD models consistently implicate gut barrier dysfunction, immuneinflammatory activation, HPA axis dysregulation and neurotransmitter imbalance as key mechanistic through-lines (Sherin and Nemeroff, 2011

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