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Therefore, we chose to use TAT peptide as the control peptide instead

NAA exerts its function in a histone acetyltransferase P300/CBP-associated factor (PCAF)-dependent manner, leading to lamin A acetylation, impairing its interaction with Sun2 (Sad1 and Unc84 domain-containing protein 2) and subsequent lytic granules, from CD8 + T cells and NK cells, polarization and release [214]

Originally identified in human plasma, GHK-Cu has been shown to regulate over 4,000 genes involved in tissue regeneration, wound healing, and inflammation control (Pickart et al., 2012)

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