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This event was, however, not necessarily preceded by an oxidative burst and later on we demonstrated that GSH release was the result of an active process that the cells carried out through specific carriers in order to efficiently execute the apoptotic program ( necessary to induce the redox unbalance characteristic of the induction phase of the mitochondrial pathway but not sufficient per se to assure the execution phase of apoptosis

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Secondary pharmacokinetic endpoints assessed maximum concentration [Cmax] of cagrilintide and semaglutide from the day of last dose (week 19) to end of treatment (week 20)

Minor injection site discomfort is possible but minimized with proper technique