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The aim of this review is to analyze the literature on the impact of GSH deficiency on the etiopathology of schizophrenia, with particular emphasis on neurodevelopmental animal models in which oxidative stress during early postnatal development causes schizophrenia-like symptoms in adulthood

doi: 10.3389/fimmu.2022.832159 50

This photothermally-enhanced approach effectively facilitates the repair of mitochondrial damage and downregulates the NF-B signaling pathway to inhibit the expression of pro-inflammatory cytokines

Primers 1 , 15056 (2015)

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