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We recently reported that low nanomolar concentration of PregS induce a delayed-onset increase of the neuronal response to NMDA and trafficking of NMDAR to the cell surface through an intracellular ([Ca 2+ ]i)-dependent mechanism (Figure 2)

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Similar to dNK cells, miR-483-3p inhibitor-transfected NK cells derived from UCB/CD34 + HSCs demonstrated upregulation of perforin and CD107a (Supplementary Fig

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