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"The role of glutathione S -transferase P in signaling pathways and S -glutathionylation in cancer"

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15 Similarly, increased generation of NAD + via the KP in resting, aged or immune-challenged macrophages restores OXPHOS and homeostatic immune responses, whereas inhibition of de novo NAD + synthesis induces an increased inflammation-associated TCA-cycle metabolite succinate and elevated mitochondria-generated ROS, resulting in rising innate immune dysfunction in aging and age-associated diseases

Oxidative Stress from Inactivation of Glutathione-Related Enzymes: A Non-Comprehensive Catalogue Inhibition or irreversible inactivation of glutathione-regulated enzymes is a key step in the derangement of cellular equilibria, possibly leading to oxidative stress

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