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Some of them are presented illustratively in the table below (Tab.1) Table 1

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Amyotrophic lateral sclerosis The molecular mechanism of excitotoxicity in ALS involves the excessive release of glutamate, causing Ca 2+ overload and excessive activation of the glutamate receptor AMPA, thereby inducing astrocyte proliferation, mitochondrial dysfunction, and energy exhaustion

I've had that exact conversation with patients who compete, and it's a hard stop for them

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