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By analyzing the reactivity of a TCR from a lesional psoriatic CD8 + T-cell clone rearranging the V3S1 and V13S1 variable region genes characteristic of infiltrating lesional CD8 + T cells (V3S1/V13S1 TCR) (5, 7), we had previously established as an underlying pathomechanism in psoriasis that HLA-C*06:02 may mediate an autoimmune response against melanocytes, and we had identified an epitope from ADAMTS-like protein 5 (ADAMTSL5) as a melanocyte autoantigen presented by HLA-C*06:02 (13, 14)

2): de novo, Preiss-Handler, and salvage (Berge et al