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In GSH-deficient mouse models of schizophrenia, neurochemical changes were observed in the cortex, including elevated levels of glutamine, glutamate, N-acetylaspartate, myo-inositol, lactate, and alanine

If the primary dysfunction is endothelial damage and impaired angiogenesis, BPC-157's VEGF pathway activation makes it the lead candidate

10.1016/j.etap.2013.05.012 4 AllenJ.BradleyR