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The cellular concentration of GSH, therefore, is regulated by a complex process of precursor amino acid transport across cell membranes, intracellular synthesizing enzymes, feedback regulation, and intracellular GSH complexing via conjugation of GSH with a variety of electrophilic compounds through GSH transferase reactions

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In uterine fibroids, dysfunction of MnSOD has been demonstrated to cause substantial accumulation of ROS and is associated with abnormal activation of the Akt signaling pathway [81]