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Pharmacological inhibition of mTORC1 suppresses excessive glycolysis and TEC proliferation, thereby ameliorating renal fibrosis (Cao et al., 2020

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This shows that the tumors intrinsic capacity to produce GSH is dispensable for its growth

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N-acetylcysteine reduces oxidative stress in sickle cell patients

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It is well documented that GSTs conjugate GSH to an array of electrophilic compounds of exogenous and endogenous origins (Cummins et al., 2011)

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