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Intriguingly, breast cancer cells in metastatic lung tumors experience higher magnitude of solid stress compared to those in the primary tumor, underscoring the key role of the local microenvironment in modulating solid stress during cancer progression [116]

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Preliminary animal and test-tube studies have noted that GHK-Cu liposomes promoted the proliferation of healthy cells, and increased the rate of scald wound healing in mice [4]

Therefore, these results suggest that a low level of PS exposure promotes axonal fusion, while a high level of PS exposure leads to axonal debris