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While CBP/p300 is a well-validated direct interaction partner of NRF2 with prominent functions at enhancers, we report that this interaction is not required for NRF2-dependent NSCLC cell growth, indicating that NRF2 can sustain sufficient transcriptional activity in the absence of CBP/p300 coactivation

Conversely, in the higher regions of the brain such as the forebrain, glycine takes on an excitatory role as an obligatory co-agonist at N-methyl-D-aspartate (NMDA) receptors

doi: 10.1152/ajpendo.90388.2008