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In addition, pharmacological interventions of FAO had obvious effects on IR injury, i.e., the activation of CPT1-dependent FAO by leptin significantly attenuated IR-induced injury, which is manifested by reduced hepatic enzyme level, hepatic injury score, hepatocyte apoptosis, and mitochondrial damage, while the inhibition of CPT1 by etomoxir imparted negative effects

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Add 2 mL of bacteriostatic water to the 10 mg vial

Therefore, the amount of copper absorbed by the intestine and retained in the body must be regulated to prevent accumulation of excess copperwhich is toxic

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