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Key molecular mechanisms include mitochondrial reverse electron transport activation induced by mutant proto-oncogenes KRAS/BRAF [385], oxidative inactivation of DNA repair enzymes leading to mutation accumulation [386], and constitutive activation of NRF2 pathway [387]

Oleic acid restores suppressive defects in tissue-resident FOXP3 Tregs from patients with multiple sclerosis

doi: 10.1186/s12967-016-1010-x 193 NaviauxRKNaviauxJCLiKBrightATAlaynickWAWangLet al

To improve the uptake efficiency of BP@CeO x by NPCs, we utilized liposome extrusion to extract NPC membrane and employed it to coat the BP@CeO x via repeated extrusion to prepare membrane-coating nanozymes (MBC)

Thus, we next analyzed if the rise of cholesterol levels in PAMP-activated microglia stimulates the secretion of neurotrophic factors