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Hyperglycemia drives intestinal barrier dysfunction and risk for enteric infection

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triagonism) on energy expenditure, glucose regulation, food intake, and weight loss in a well-established DIO mouse model in comparison to clinically relevant GLP1 receptor agonists [83]

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The combination leverages complementary mechanisms: amylin primarily delays gastric emptying and signals satiety through the area postrema, while GLP1 enhances pancreatic cell function and central appetite suppression [1]

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Probiotic therapy in Helicobacter pylori infection: a potential strategy against a serious pathogen

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