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These results encouraged the possibility that a clinically useful drug can be designed possessing oral efficacy, BBB penetrability, increased metabolic stability, coupled with facilitated cognitive functioning

Simultaneously, leaked electrons combine with oxygen molecules, causing bursts of mitochondrial-derived ROS, which have become a significant source of oxidative stress in cells (50, 51)

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The mechanism centers on TREK-1, a two-pore domain potassium (K2P) channel widely expressed in the central nervous system