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At the molecular level, BPC-157 activates VEGFR2 signaling to drive new vessel formation at wound sites, mobilizes FAK-paxillin complexes that govern cell adhesion and directional migration, and upregulates growth hormone receptor expression in fibroblasts dramatically amplifying the healing cascade

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Seung-Chul Choi, The University of Texas Health Science Center at San Antonio, United States

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Zhang L, Kang H, Zhang W, Wang J, Liu Z, Jing J, et al