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In conclusion, AMSC-based cell therapy restores mitochondrial function in APAP-induced hepatotoxicity via the DDIT4/PGC-1 pathway in vivo

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Ca 2+ homeostasis imbalance Upon the release of the excitatory neurotransmitter glutamate from the presynaptic terminal, it initiates the activation of postsynaptic receptors, thereby inducing alterations in the electrical potential across the postsynaptic membrane

The majority of preclinical musculoskeletal research on BPC-157 used injectable administration - subcutaneous or intramuscular delivery in animal models