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Numerous studies have shown the wide application of c-Met in digestive system cancers with related clinical and preclinical evidences supporting the anti-HGF/c-Met signaling pathway as a reliable treatment for HCC, gastric, pancreatic, and CRC among others

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Crucially, its enhanced metabolic stability and superior bioavailability enable it to readily traverse the blood-brain barrier, surpassing the therapeutic limitations of its parent molecule