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So here's what I would suggest

To date there is little information on the functioning and copper loading of the membrane-bound CP found in cells of the CNS in AD, which would be useful to determine (Ward et al., 2014)

Large doses of folic acid may also partially mask or worsen vitamin B deficiency by correcting anemia while allowing neurological damage to progress

Since no appropriate PR3-AAV model exists, clinical studies with adjunctive DPP1 inhibitor administration in patients with PR3-AAV may provide the best approach to test this hypothesis

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