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Turkovic B, Sikiric P, Seiwerth S, et al

[DOI] [PMC free article] [PubMed] [Google Scholar] 17.Rajakovich LJ, Fu B, Bollenbach M, Balskus EP

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Additionally, obesity and IR both contribute to -cell dysfunction and apoptosis, mechanistically explained by glucotoxicity and increased immunogenicity (the accelerator hypothesis) or by -cell exhaustion due to increased metabolic demand (the overload hypothesis) [23]