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2.3 Free radical scavenging capacity of Ru SA+AC on oxidative stress rat chondrocytes in vitro An overabundance of RONS can lead to increased expression of inflammatory factors, mitochondrial impairment, and the induction of cell apoptosis, thereby accelerating the progression of OA[64]
Loss of SHMT2 activity is accompanied by compensatory reversal catalyzed by SHMT1, along with the synthesis of 1C units and glycine from serine
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*Grow should be cycled
