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Overexpression of normal human Ataxin-3 can protect Drosophila from polyQ-huntingtin-induced neurodegeneration (Warrick et al., 2005)

Acylase I-catalyzed deacetylation of N-acetyl-L-cysteine and S-alkyl-N-acetyl-L-cysteines

Dihexa was originally developed as a potential treatment for Alzheimers disease and dementia, aiming to restore lost neural connections

It creates a permanent upstream bottleneck that compounds existing enzyme-level impairments in glutathione production