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In neuronal senescence model studies, the active ingredient of the product can cross the blood-brain barrier (and act directly on neurons in vitro), increase NAD+ levels in nerve cells, protect mitochondrial function, reduce neuronal apoptosis, and promote the synthesis and release of neurotransmitters (such as dopamine and acetylcholine), thereby improving signal transduction in nerve cells

Ferroptosis in RM is driven by dysregulated iron metabolism and increased oxidative stress, resulting from impaired antioxidant defense, which leads to lipid peroxidation and consequent cell death in trophoblasts

Qureshi, I

Comparative proteomic approaches for the isolation of proteins interacting with thioredoxin

Table 2 aims to yield a direct comparison between various nanoformulations and address BBB transport mechanism, toxicity profile along with translational considerations