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In addition, A deposition and excessive tau phosphorylation in the brains of AD patients can further inhibit the expression of SLC7A11, exacerbating GSH depletion and GPX4 inactivation, and forming a vicious cycle of oxidative stress-ferroptosis-AD pathological damage

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Supplementary material The Supplementary Material for this article can be found online at: References 1 BaoY

FDA Status: As of 2026, the FDA has not approved BPC-157 for human consumption