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Research into whether modified lactococcus lactis bacteria can transport BPC-157 to the gastrointestinal tract shows that the bacteria dramatically increase levels of the peptide in cell culture

It could alter how the peptide folds, how it binds to receptors, its overall stability, or its mechanism of action

Instead it works through a distinct set of mechanisms: NFB inhibition: KPV's primary action is blocking NFB, the master switch that drives inflammatory gene expression in immune and epithelial cells

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