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According to the proposed mechanism, Gln consumption by ccRCC tumor cells leads to local depletion of extracellular matrix Gln, thereby activating the HIF1/IL-23 signaling pathway in tumor-infiltrating macrophages

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Enzyme mimics include metal-based complexes and nanomaterials (Ren et al

TGR inhibitors do not react covalently with GSH, selenocysteine, or TGR, and inhibition of TGR is reversible To evaluate stability of the TGR inhibitors in the presence of GSH and selenocysteine, compound 2 and a known covalent inhibitor of TGR, TRi-1 20,45 , were incubated with GSH or the N-Boc protected methyl ester of selenocysteine, and the reaction mixtures were analyzed by LCMS (Supplementary Figs

doi: 10.1002/ardp.202300569 46 NygaardGFiresteinGS