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In summary, our data implicate a novel mechanism protecting against liver steatosis via an oxidative stress adaptive response that activates the AMPK pathway

GHK-Cu, by contrast, appears to act more directly on fibroblast signaling and collagen gene transcription, with VEGF upregulation occurring as part of a broader tissue remodeling response

No tolerance development has been documented, suggesting ongoing benefits with consistent use

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[DOI] [PMC free article] [PubMed] [Google Scholar] 278.Udovicic M., Sever M., Kavur L., Loncaric K., Barisic I., Balenovic D., Zivanovic Posilovic G., Strinic D., Uzun S., Batelja Vuletic L., et al