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In all cell models analyzed, tivantinib did not inhibit HGF-dependent or HGF-independent c-Met tyrosine autophosphorylation [85]

glutamylcysteinylglycine) is the principal lowmolecular-weight thiol buffer in mammalian cells (millimolar, largely reduced), maintaining redox poise and integrating detoxification with redox-sensitive signaling

35 are preclinical, and one is on a human trial, and so it was very limited in human research, so we got a lot of rats and dogs that were utilized for these studies

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