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10.1038/s41589-021-00962-9

Lack of critical involvement of caspase-3 in eryptosis has been emphasized in an extensive analysis of the caspase-3 contribution to eryptosis, which has revealed that eryptosis triggered in vitro by diverse compounds is not associated with caspase-3 recruitment in the majority of cases [84]

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Both P53 and P16INK4a prevent the activation of CDK4/6, leading to the inhibition of retinoblastoma protein (pRB) phosphorylation and subsequent cell cycle arrest in the G1 phase [203]