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Suppression of the malignant phenotype in human pancreatic cancer cells by the overexpression of manganese superoxide dismutase

Conversely, Fsp1 knockout in MTECs had the opposite effects

These will increase glutathione levels in the body which can likewise render the treatments ineffective

Examples include the upregulation of cyclic adenosine monophosphate-responsive element binding protein-regulated transcription coactivator (CRTC) 2 and phosphorylated acetyl-CoA carboxylase (p-ACC), or downregulation of peroxisome proliferator-activated receptor (PPAR) , acetyl-CoA carboxylase (ACC), and sterol regulatory element binding protein 1 (SREBP1) (63)

In addition, recent advances in ferroptosisrelated biomarkers and therapeutic strategies are highlighted, with the aim of providing a clearer framework for understanding its pathogenic roles and guiding future clinical translation