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HFs are specialized organelles that are constantly recycling

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Beyond the established mechanisms, a pivotal contributor to DCM pathogenesis is the disruption of systemic and cellular copper homeostasis, which primarily inflicts damage through the aberrant compartmentalization of copper within cardiac tissues [80]

It accomplishes this through several distinct mechanisms: Collagen and elastin stimulation: GHK-Cu upregulates the genes responsible for collagen I, collagen III, and elastin synthesis the structural proteins that give skin its firmness, bounce, and thickness