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Initial research focused on its role in wound healing and tissue remodeling, where it was found to modulate the expression of extracellular matrix (ECM) proteins such as collagen and elastin

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Endpoint Assessment: Lipolysis: Glycerol and fatty acid release Gene expression: Lipid metabolism enzymes Body composition: Fat and lean mass Metabolic parameters: Without glucose/IGF-1 changes Mechanism Investigation: Key research questions: What is the molecular target of AOD9604

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Increased PPAR degradation inhibited downstream fatty acid metabolism, leading to renal lipid accumulation, disrupted energy metabolism, and impaired kidney function