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Review of molecular mechanisms involved in the activation of the Nrf2-ARE signaling pathway by chemopreventive agents

I is a kind of reducing agent that can be oxidized by peroxidase enzymes to generate thyroid hormones (THs)

These responses including glial activation, metabolic braking, and suppression of plasticity-related signaling are biologically adaptive in the short term, but may become limiting when they remain chronically engaged

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A critical future research direction should involve optimizing combination therapy regimens, defining the optimal timing and dosing strategies, and developing specific targeting modalities that minimize off-target effects