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As a bioactive metabolite, TMAO can activate various intracellular signaling cascades, including the MAPK and NF-B pathways, the NLRP3 inflammasome, and PERK signaling as wellmechanisms closely linked to chronic inflammation (Figures 2, 3, and 4)

Furthermore, the same receptor expressed in two distinct cell types can be endocytosed through different mechanisms due to differences in GRK and arrestin expression: either through clathrin-coated vesicles or caveolae

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Physiol Plant 77:465471 CrossRefGoogle Scholar S -transferase inhibitor overcomes the P-glycoprotein-mediated resistance in tumor cells: 6-(7-nitro-2, 1, 3-benzoxadiazol-4-ylthio) hexanol (NBDHEX) triggers a caspase-dependent apoptosis in MDR1-expressing leukemia cells

The scRNA-seq data were aligned to the Ensembl_v105_mus_musculus and row count matrices were imported to the R package Seurat V3.1.1