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Induction of c-Jun requires activation of c-Jun N-terminal kinase (JNK) through the phosphorylation on its Serine 63 and 73 residues

Oxidative distress causes damage to fundamental macromolecules including proteins, lipids, RNA and DNA at a cellular level, which promotes abnormal cell death and inflammation, often culminating in additional oxidative stress at a systemic level

In diabetic rats, which are known to exhibit severely delayed wound healing, PIC treatment demonstrated elevated glutathione and ascorbic acid levels, improved epithelialization, and enhanced activation of fibroblasts and mast cells in the wound area

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8 weeks (3 vials): ~9 mL 1 bottle 16 weeks (6 vials): ~18 mL 2 bottles Alcohol Swabs One for the vial stopper + one for the injection site each day