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In preclinical models it has been associated with cytoprotection , modulation of nitric-oxide pathways and angiogenesis , with reported activity across gastrointestinal, musculoskeletal and other tissues [1] [2]

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

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Causative mechanisms for CV-PASC include chronic inflammation due to viral persistence in heart tissue, molecular mimicry invoking autoimmune responses against cardiac antigens, and ongoing endothelial/microvascular dysfunction 192

Champier, J., Claustrat, F., Nazaret, N., Fevre Montange, M