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Regulatory note: Injectable glutathione is not FDA-approved

490 Excessive inhibition of Nrf2 may not improve DOX-induced cardiomyopathy due to insufficient promotion of antioxidant gene expression, while its overactivation increases the breakdown of heme and ferrous iron levels in the HO-1-catalyzed manner, promoting ferroptosis

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This heightened vulnerability becomes more evident when there is damage to the diabetic heart, ventricular remodeling, and heart failure, where therapeutic strategies targeting ferroptosis could offer promising benefits

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