As confirmation, we used the crisis that occurred with the high intra-abdominal pressure-induced syndrome, in which intra-abdominal hypertension simultaneously affected all abdominal vessels and organs for a considerable period and restrained the ability to recruit alternative pathways, such that a deadly situation was created before therapy initiation
At the same time, it appeared that some transcript readouts moved in different directions, which suggests that FOXO4-DRI peptide may also act as a stressor for surviving cells, potentially triggering compensatory transcriptional responses even as senescent cells are eliminated
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Complementary approaches like surface charge neutralization, PEGylation modifications, or Fc-region fusion could further enhance serum half-life while mitigating ADA responses 36,37
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