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This, therefore, begs the question, what happens if these instructions are received by a cell which is already damaged, pre-cancerous or on the edge

The complexity increases but remains manageable with proper planning

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If binding of one IGF1 molecule to the IGF1R dimer is sufficient for receptor activation, then both the WT:WT and WT:F701A dimers are expected to be active, and the IGF1R activity in cells transfected with WT and F701A plasmids will be 75% of that in cells transfected with the WT plasmid alone

However, sourcing, dosage, and administration are everything